Mitochondrial dysfunction is a key factor in the development of atherogenesis. METTL4 (methyltransferase-like protein 4) mediates N6- methyldeoxyadenosine (6mA) of mammalian mitochondrial DNA (mtDNA).
The changes persist after AAS withdrawal, and normalisation depends on the duration of the drug abuse. Eight weeks of administration of nandrolone decanoate does not affect lipid and lipoprotein ...
In this Review, Borén and colleagues present insights into the role of apolipoprotein B-containing lipoproteins in atherogenesis, with an emphasis on the mechanisms leading to atherosclerotic ...
There are striking differences in distribution of lipoproteins among the different ages and sexes. During the stages of active atherogenesis in hypertensives and nephrotics, both the α and β ...
[1] E. C. Rizos, E. E. Ntzani, E. Bika, M. S. Kostapanos and M. S. Elisaf, “Association between Omega-3 Fatty Acid Supplementation and Risk of Major Cardiovascular ...
Another complicating factor in the early assessment of the premature infant is the fluctuation or transient nature of muscle tone. The incidence of transient neurologic abnormalities ranges from ...